Caffeine Consumption and Induction of Obesity in the Developed World

Editorial

Ann Obes Disord. 2017; 2(1): 1018.

Caffeine Consumption and Induction of Obesity in the Developed World

Martins IJ1,2,3*

¹School of Medical Sciences, Edith Cowan University, Australia

²School of Psychiatry and Clinical Neurosciences, University of Western Australia, Australia

³McCusker Alzheimer’s Research Foundation, Hollywood Medical Centre, Australia

*Corresponding author: Ian James Martins, School of Medical Sciences, Edith Cowan University, 270 Joondalup Drive, Joondalup, Western Australia 6027, Australia

Received: January 13, 2017; Accepted: January 19, 2017; Published: January 23, 2017

Keywords

Caffeine; Appetite; Obesity; Adipose tissue; Non alcoholic fatty liver disease; Magnesium

Editorial

The susceptibility of humans to obesity favors the deposition of fat with fat deposition in man much higher than compared with other species. The global increase in Non Alcoholic Fatty Liver Disease (NAFLD) is linked to the obesity epidemic and appetite dysregulation [1-3] is possibly the major defect in man with over eating the major cause of adiposity and NAFLD. Environment and genes have been indicated to play a major role in the global obesity epidemic with alterations in the appetite centre early in life relevant to the defective adipose tissue-liver crosstalk [4]. Induction of NAFLD may favor adipogenesis with consumption of high calorie diets, lack of essential nutrients, palmitic acid rich diets, nuclear receptor inhibitors, magnesium deficiency and bacterial lipopolysaccharides all relevant to liver inflammation and steatosis (Figure 1).

Citation: Martins IJ. Caffeine Consumption and Induction of Obesity in the Developed World. Ann Obes Disord. 2017; 2(1): 1018.